These are exposure studies associated with the chemical and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Arbuckle TE, et al. (2015).||Maternal-Infant Research on Environmental Chemicals (MIREC) Study||We report national-level data on urinary concentrations of free and conjugated forms of bisphenol A and triclosan in pregnant women and suggest that maternal characteristics predicting elevated urinary concentrations of these phenols largely act in opposite directions.||age | socioeconomic status | tobacco||bisphenol A | Triclosan||Pregnant females||Canada||urine||bisphenol A | bisphenol A disulfate | bisphenol A glucuronide | Triclosan||Details|
|2.||Valentine GW, et al. (2016).||Brief use of a widely available type of e-cigarette containing an e-liquid purchased from an Internet vendor can negatively impact psychomotor performance and in some instances, produce detectable levels of a urine alcohol metabolite.||Ethanol | Nicotine||Study subjects||United States||urine||ethyl glucuronide||Details||psychomotor behavior|
|3.||Peters KO, et al. (2017).||Center for Childhood Asthma in the Urban Environment (CCAUE)||Time spent in non-smoking homes was associated with significantly decreased urine 1-hydroxypyrene-glucuronide (1-OHPG), and secondhand smoke exposures increased these levels; time spent outdoors was associated with increased urinary 1-OHPG concentrations in boys only; our results suggest that secondhand smoke and ambient outdoor air pollution contribute to internal dose of polycyclic aromatic hydrocarbons in inner city children.||sex | tobacco||Particulate Matter | Polycyclic Aromatic Hydrocarbons | Tobacco Smoke Pollution||Children||United States||air, indoor | urine||1-hydroxypyrene-glucuronide | Nicotine | Particulate Matter||Details|