These are exposure studies associated with the chemical and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Delfino RJ, et al. (2010).||Traffic emission sources of organic chemicals represented by polycyclic aromatic hydrocarbons are associated with increased systemic inflammation and explain associations with quasi-ultrafine particle mass.||Air Pollutants | hopane | Polycyclic Aromatic Hydrocarbons | Vehicle Emissions||Subjects with disease:Coronary Artery Disease||United States||air, outdoor | plasma||Alkanes | Chromium | Copper | hopane | IL6 | Iron | Manganese | Nickel | Particulate Matter | Polycyclic Aromatic Hydrocarbons | TNFRSF1B | Vanadium | Zinc||Details|
|2.||Sbihi H, et al. (2013).||Canadian Healthy Infant Longitudinal Development (CHILD) | Toronto Child Health Evaluation Questionnaire (TCHEQ) | Windsor Ontario Exposure Assessment Study (WOEAS)||Hopanes can consistently be detected in house dust and may be a useful tracer of traffic-related air pollution exposure if determinants of their spatiotemporal variability are well-characterized, and when home-specific factors are considered.||hopane||Study subjects||Canada||dust||hopane||Details|