These are exposure studies associated with the chemical and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Johansson GM, et al. (2015).||This first study of amine adducts in hairdressers and consumers identified an increase in o- and m-toluidine Hb-adduct concentrations among hairdressers, associated with the weekly number of hair dye and hair waving treatments performed.||2,3-
||Study subjects | Workers||Sweden||blood||2,3-
|2.||Adad LM, et al. (2015).||In this study, we determined the mutagenic and cell death potential of pesticides through the increased frequency of micronuclei and other nuclear abnormalities in exposed individuals||abamectin | copper oxychloride | Dimethoate | glyphosate | Methyl Parathion | Triazines||Workers||Brazil||Details||cholinesterase activity | cytokinetic process | lipid metabolic process | micronucleus organization | nuclear fragmentation involved in apoptotic nuclear change|
|3.||Adgent MA, et al. (2015).||National Health and Nutrition Examination Survey (NHANES)||Antibiotics, but not triclosan, are negatively associated with urinary enterolactone; further study of triclosan exposure and enterolactone production may be needed to better understand positive associations among women.||age | body mass index | diet | race | sex | socioeconomic status||Anti-Bacterial Agents | Lincomycin | Macrolides | Quinolones | Sulfonamides | Triclosan||Study subjects||United States||urine||2,3-
|4.||Paterson JM, et al. (2012).||Among older outpatients with no evidence of liver disease, moxifloxacin and levofloxacin were associated with an increased risk of acute liver injury relative to clarithromycin.||cefuroxime axetil | Ciprofloxacin | Clarithromycin | Levofloxacin | moxifloxacin||Controls for disease:Chemical and Drug Induced Liver Injury | Subjects with disease:Chemical and Drug Induced Liver Injury||Canada||Details||Chemical and Drug Induced Liver Injury|