These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Andersen ZJ, et al. (2012).||Danish Diet, Cancer, and Health cohort Study||Long-term exposure to traffic-related air pollution may contribute to the development of ischemic but not hemorrhagic stroke, especially severe ischemic strokes leading to death within 30 days.||Air Pollutants||Study subjects||Denmark||Nitrogen Dioxide||Details||Brain Ischemia | Cerebral Hemorrhage | Stroke|
|2.||Shcherbatykh I, et al. (2005).||These results suggest that living near a source of persistent organic pollutants contamination constitutes a risk of exposure and an increased risk of acquiring cerebrovascular disease.||age | race | sex||Dioxins | Pesticides | Polychlorinated Biphenyls||Study subjects||United States||Details||Cerebral Infarction|
|3.||O'Donnell MJ, et al. (2011).||These results do not support the hypothesis that short-term increases in particulate matter PM2.5 levels are associated with ischemic stroke risk overall. However, specific patient subgroups (e.g., diabetes mellitus) may be at increased risk of particulate-related ischemic strokes.||disease||Particulate Matter||Subjects with disease:Diabetes Mellitus||Canada||air, ambient||Particulate Matter||Details||Ischemic Attack, Transient | Stroke|
|4.||Chen WH, et al. (2006).||A chemical worker working with urea-formaldehyde resin hazard for 20 years suffered cerebral ischemia in association with an increase of anticardiolipin antibody and a prolongation of activated partial thromboplastin time.||urea formaldehyde foam||Workers||Taiwan, Province of China||Details||Brain Ischemia | Thrombosis | humoral immune response mediated by circulating immunoglobulin | immunoglobulin production involved in immunoglobulin mediated immune response|
|5.||Mao X, et al. (2014).||We describe associations among basic characteristics, seasons, and diseases with serum levels of 25-hydroxyvitamin D in patients and note that 25(OH)D3 levels are low while 25(OH)D2 levels are high among patients with lung diseases, dyskinesias, and coronary heart disease, and participants with diabetes and cerebral infarction have higher 25(OH)D3 serum concentrations compared with lung disease patients.||age | disease||Vitamin D||Subjects with disease:Cerebral Infarction | Subjects with disease:Coronary Disease | Subjects with disease:Diabetes Mellitus | Subjects with disease:Dyskinesias | Subjects with disease:Hypertension | Subjects with disease:Lung Diseases | Study subjects||China||serum||25-Hydroxyvitamin D 2 | 25-hydroxyvitamin D | Calcifediol||Details|
|6.||Majumdar V, et al. (2015).||The relationship between vitamin D deficiency and stroke was cross-sectionally evaluated in the high-risk Asian Indian population, and we conclude that hypertension partly explains the association between low 25-hydroxyvitamin D levels and ischemic stroke.||disease||Vitamin D||Controls for disease:Cerebral Infarction | Subjects with disease:Cerebral Infarction | Subjects with disease:Hypertension||India||serum||25-hydroxyvitamin D||Details||Cerebral Infarction|