These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Brough HA, et al. (2015).||Consortium of Food Allergy Research Observational Study (CoFAR)||Exposure to peanut antigen in dust through an impaired skin barrier in atopically inflamed skin is a plausible route for peanut skin sensitization and peanut allergy.||diet | disease||Antigens, Plant||Children | Subjects with disease:Dermatitis, Atopic||United States||dust||Antigens, Plant||Details||Peanut Hypersensitivity|
|2.||Kim HH, et al. (2016).||The present study showed that traffic-related and other pollutants around primary school areas are associated with increased risks of allergic diseases among elementary school students.||Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Soot | Sulfur Dioxide||Children||Korea, Republic of||air||Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Soot | Sulfur Dioxide||Details||Asthma | Dermatitis, Atopic | Rhinitis, Allergic|
|3.||Standl M, et al. (2011).||German Infant Study on the Influence of Nutrition Intervention plus Environmental and Genetic Influences on Allergy (GINIplus) | Lifestyle-Related factors on the Immune System and the Development of Allergies in Childhood plus the influence of traffic emissions and genetics (LISAplus)||The association between dietary intake of fatty acids (e.g., margarine) and allergic diseases (e.g., asthma) in children might be modulated by variants in the fatty acid desaturase gene cluster.||diet | genetics||Margarine||Controls for disease:Asthma | Subjects with disease:Asthma | Children | Controls for disease:Eczema | Subjects with disease:Eczema | Subjects with gene influence:FADS1 | Subjects with gene influence:FADS2 | Controls for disease:Ige Responsiveness, Atopic | Subjects with disease:Ige Responsiveness, Atopic | Controls for disease:Rhinitis, Allergic, Seasonal | Subjects with disease:Rhinitis, Allergic, Seasonal||Germany||margarine||Margarine||Details||Asthma|
|4.||Grandjean P, et al. (2010).||These findings suggest that developmental exposure to immunotoxicants may both increase and decrease the risk of allergic disease and that associations between breast-feeding and subsequent allergic disease in children may, at least in part, reflect lactational exposure to immunotoxic food contaminants.||Mercury | Polychlorinated Biphenyls||Children | Mothers | Pregnant females||Faroe Islands||blood | blood, cord | hair | milk, human | serum||Mercury | Polychlorinated Biphenyls||Details||Asthma | Dermatitis, Atopic | immunoglobulin production|
|5.||Czarnobilska E, et al. (2012).||Altogether, these observations demonstrate that the rates of contact sensitizations in children reflect changes in their environment, and limitations imposed on the use of haptens with strong sensitizing properties, may be an effective tool in the prevention of contact allergy.||Nickel | Peruvian balsam | Propolis | Thimerosal||Children||Poland||Nickel | Peruvian balsam | Propolis | Thimerosal||Details||Dermatitis, Allergic Contact|
|6.||Kponee KZ, et al. (2015).||In this cross-sectional pilot study, the first carried out in response to the United Nations Environment Programme recommendations, we observed statistically significant associations between exposure to petroleum-contaminated drinking water and self-reported symptoms consistent with exposure to petroleum hydrocarbons.||Petroleum | Water Pollutants, Chemical||Study subjects||Nigeria||Details||Anemia | Dermatitis, Irritant | Dizziness | Eye Pain | Headache | Pharyngitis|