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Disease Nervous System Diseases

These are exposure studies associated with the disease and all of its children.

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1–50 of 111 results.
  Reference Associated Study Title Author's Summary Study Factors Stressor Receptors Country Medium Exposure Marker Measurements Outcome
1. Malek AM, et al. (2015). A potential association was found for exposure to ambient air concentrations of suspected neurotoxicant hazardous air pollutants, specifically aromatic solvents, among place of residence in 2002 and 1999 and risk of Amyotrophic Lateral Sclerosis. 1,1,1-trichloroethane | 1,1,2,2-tetrachloroethane | 2,4-dinitrotoluene | Acrylamide | allyl chloride | Arsenicals | Benzene | Cadmium Compounds | Carbon Disulfide | Carbon Tetrachloride | Chloroform | Cresols | Cyanides | ethylbenzene | Ethylene Dibromide | ethylene dichloride | Ethylene Oxide | Hexachlorobenzene | hexachloroethane | Hexanes | hydrazine | Lead | Manganese Compounds | Mercury Compounds | Methyl Chloride | Methylene Chloride | Nickel | Polychlorinated Biphenyls | Selenium Compounds | Styrene | Tetrachloroethylene | Toluene | Trichloroethylene | Vinyl Chloride | Xylenes Controls for disease:Amyotrophic Lateral Sclerosis | Subjects with disease:Amyotrophic Lateral Sclerosis United States 1,1,1-trichloroethane | 1,1,2,2-tetrachloroethane | 2,4-dinitrotoluene | Acrylamide | allyl chloride | Arsenicals | Benzene | Cadmium Compounds | Carbon Disulfide | Carbon Tetrachloride | Chloroform | Cresols | Cyanides | ethylbenzene | Ethylene Dibromide | ethylene dichloride | Ethylene Oxide | Hexachlorobenzene | hexachloroethane | Hexanes | hydrazine | Lead | Manganese Compounds | Mercury Compounds | Methyl Chloride | Methylene Chloride | Nickel | Polychlorinated Biphenyls | Selenium Compounds | Styrene | Tetrachloroethylene | Toluene | Trichloroethylene | Vinyl Chloride | Xylenes Details Amyotrophic Lateral Sclerosis
2. Mi T, et al. (2013). We describe two cases of acute toxic leukoencephalopathy in migrant workers who were occupationally exposed to organic solvents in construction materials. 1,2,3-trichloropropane | Solvents | Toluene Workers China blood | urine 1,2,3-trichloropropane | Benzoic Acid Details Leukoencephalopathies | Neurotoxicity Syndromes | Occupational Diseases
3. Rull RP, et al. (2006). Data on two case-control study populations of infants with neural tube defects (NTDs) and nonmalformed controls delivered in California between 1987 and 1991 were pooled to investigate whether maternal residential proximity to applications of specific pesticides or physicochemical groups of pesticides during early gestation increases the risk of these malformations. 1,3-dichloro-1-propene | Amides | Benomyl | Benzimidazoles | Captan | Chlorpyrifos | devrinol | Dimethoate | Disulfoton | Methomyl | methyl carbamate | methyl demeton | Naled | Organophosphorus Compounds | vendex Infants or newborns United States 1,3-dichloro-1-propene | Amides | Benomyl | Benzimidazoles | Captan | Chlorpyrifos | devrinol | Dimethoate | Disulfoton | Methomyl | methyl carbamate | methyl demeton | Naled | Organophosphorus Compounds | vendex Details Anencephaly | Neural Tube Defects | Spinal Dysraphism
4. Lyall K, et al. (2017). Early Markers for Autism (EMA) | Project Baby's Breath (PBB) The overall pattern of our results suggests increases in risk of autism spectrum disorder and intellectual disability without autism with prenatal exposure to higher levels of a number of organochlorine compounds, particularly polychlorinated biphenyl ethers. 2,2',3,3',4,4',5-heptachlorobiphenyl | 2,2',4,4',5-pentachlorobiphenyl | 2,3',4,4',5-pentachlorobiphenyl | 2,4,4'-trichlorobiphenyl | 2,4,5,2',4',5'-hexachlorobiphenyl | Dichlorodiphenyl Dichloroethylene | nonachlor | PCB 180 | Polychlorinated Biphenyls Controls for disease:Autism Spectrum Disorder | Subjects with disease:Autism Spectrum Disorder | Children | Controls for disease:Intellectual Disability | Subjects with disease:Intellectual Disability | Pregnant females United States serum 2,2',3,3',4,4',5-heptachlorobiphenyl | 2,2',4,4',5-pentachlorobiphenyl | 2,3',4,4',5-pentachlorobiphenyl | 2,4,4'-trichlorobiphenyl | 2,4,5,2',4',5'-hexachlorobiphenyl | Dichlorodiphenyl Dichloroethylene | nonachlor | PCB 180 | Polychlorinated Biphenyls Details Autism Spectrum Disorder | Intellectual Disability | Prenatal Exposure Delayed Effects
5. Herbstman JB, et al. (2010). This epidemiologic study demonstrates neurodevelopmental effects in relation to cord blood polybrominated diphenyl ether concentrations. 2,2',4,4',5,6'-hexabromodiphenyl ether | Halogenated Diphenyl Ethers | pentabromodiphenyl ether Infants or newborns United States blood, cord 2,2',3,4,4',5',6-heptabromodiphenyl ether | 2,2',4,4'-tetrabromodiphenyl ether | hexabrominated diphenyl ether 153 | hexabromodiphenyl ether 154 | PBB 153 | pentabromodiphenyl ether Details Intellectual Disability | Psychomotor Disorders | cognition
6. Kamel F, et al. (2007). Agricultural Health Study (AHS) This study suggests that exposure to certain pesticides may increase Parkinson's disease risk. 2,4,5-Trichlorophenoxyacetic Acid | Benomyl | butylate | Carbon Disulfide | Carbon Tetrachloride | cyanazine | Dicamba | Ethylene Dibromide | Lindane | mancozeb | Maneb | methyl bromide | Paraquat | pendimethalin | Pesticides | Phorate | tetrachloroisophthalonitrile Study subjects | Workers United States Details Agricultural Workers' Diseases | Parkinson Disease
7. Marfo JT, et al. (2015). Urinary N-desmethylacetamiprid can be used as a biomarker for environmental exposure to acetamiprid, and detection of N-desmethylacetamiprid was associated with increased prevalence of neurological symptoms. acetamiprid | Insecticides Controls for disease:Neurologic Manifestations | Subjects with disease:Neurologic Manifestations Japan urine ((E)-1-(2-chloro-1,3-thiazol-5-ylmethyl)-3-methyl-2-nitroguanidine) | N-desmethylacetamiprid | nitenpyram | thiacloprid | thiamethoxam Details Neurologic Manifestations
8. Beard JD, et al. (2016). Genes and Environmental Exposures in Veterans with Amyotrophic Lateral Sclerosis (GENEVA) We evaluated associations between military-related factors and amyotrophic lateral sclerosis using data from a case-control study of US military veterans; among some of the positive associations (needing confirmation) were exposure to military herbicides, military nasopharyngeal radium, and Agent Orange. Agent Orange | Defoliants, Chemical | Radium Subjects with disease:Amyotrophic Lateral Sclerosis | Veterans United States Details Amyotrophic Lateral Sclerosis
9. Andersen ZJ, et al. (2012). Danish Diet, Cancer, and Health cohort Study Long-term exposure to traffic-related air pollution may contribute to the development of ischemic but not hemorrhagic stroke, especially severe ischemic strokes leading to death within 30 days. Air Pollutants Study subjects Denmark Nitrogen Dioxide Details Brain Ischemia | Cerebral Hemorrhage | Stroke
10. Liu R, et al. (2016). Parkinson's Genes and Environment Study (PAGE) Overall, we found limited evidence for an association between exposures to ambient particulate matter PM10, PM2.5, or nitrogen dioxide and Parkinson disease risk; however, the suggestive evidence that exposures to PM2.5 and PM10 may increase disease risk among female never-smokers warrants further investigation. sex | tobacco Air Pollutants Controls for disease:Parkinson Disease | Subjects with disease:Parkinson Disease United States air, ambient Nitrogen Dioxide | Particulate Matter Details Parkinson Disease
11. Zhang P, et al. (2011). Long-term exposure to ambient air pollution is associated with the death of cardiovascular and cerebrovascular diseases among Chinese populations. The effects of air pollution were more evident in female that in male, and nonsmokers and residents with body mass index less than 18.5 were more vulnerable to outdoor air pollution. body mass index | sex | tobacco Air Pollutants Study subjects China air, ambient Nitrogen Dioxide | Particulate Matter | Sulfur Dioxide Details Cardiovascular Diseases | Cerebrovascular Disorders | Death
12. Lin CC, et al. (2014). Taiwan Birth Cohort Pilot Study (TBCS-p) Low-level sulfur dioxide exposure prenatally and up to 12 months postnatal could cause adverse neurobehavioral effects at 18 months of age; furthermore, adverse gross motor below average scores at 6 months of age were associated with increased non-methane hydrocarbon exposure during the second and third trimesters. Air Pollutants Fetuses | Infants or newborns | Pregnant females Taiwan, Province of China air, ambient Carbon Monoxide | Hydrocarbons | Nitrogen Dioxide | Ozone | Particulate Matter | Sulfur Dioxide Details Neurobehavioral Manifestations | Prenatal Exposure Delayed Effects | motor behavior
13. Li Z, et al. (2011). Compared with women with no indoor air pollution from coal combustion exposure, women with any exposure at all had a 60% increased risk of having a child with an neural tube defects. An increased risk was linked to both residential heating and cooking. This is the first known study to link indoor air pollution from coal combustion to neural tube defects. Air Pollutants | Coal | Smoke Study subjects China Details Neural Tube Defects
14. Raaschou-Nielsen O, et al. (2011). Diet, Cancer and Health Study This hypothesis-generating study indicates that traffic-related air pollution might increase the risks for cervical and brain cancer, which should be tested in future studies. Air Pollutants | Vehicle Emissions Study subjects Denmark air Nitrogen Oxides Details Brain Neoplasms | Uterine Cervical Neoplasms
15. Zhang W, et al. (1994). We report early health effects and biological monitoring in persons occupationally exposed to tetraethyl lead in China for the years 1990-1992, including for gasoline depot workers and traffic police officers. Air Pollutants, Occupational | Tetraethyl Lead Workers China air, ambient | urine diethyllead | Lead | triethyllead Details Bradycardia | Tremor
16. Starks SE, et al. (2012). Agricultural Health Study (AHS) This study provides some evidence that long-term exposure to organophosphate pesticides is associated with signs of impaired peripheral nervous system function among pesticide applicators. Aldicarb | Chlorpyrifos | Coumaphos | Dichlorvos | Dimethoate | Disulfoton | ethoprop | Fonofos | Phosmet | phostebupirim | Tetrachlorvinphos Workers United States Details Agricultural Workers' Diseases | Gait Disorders, Neurologic | Neurologic Manifestations | Reflex, Abnormal | Somatosensory Disorders | Tremor
17. Kamel F, et al. (2012). Agricultural Health Study (AHS) The meta-analysis suggests that amyotrophic lateral sclerosis risk is associated with use of pesticides as a group, and our analysis of Agricultural Health Study data points to organochlorine insecticide use in particular. Aldrin | DDT | Dieldrin | Herbicides | Hydrocarbons, Chlorinated | Pesticides | Pyrethrins | Toxaphene Study subjects | Workers United States Details Agricultural Workers' Diseases | Amyotrophic Lateral Sclerosis
18. Kamel F, et al. (2014). Agricultural Health Study (AHS) | Farming and Movement Evaluation Study (FAME) Polyunsaturated fatty acid intake was consistently associated with lower Parkinson's disease (PD) risk, and dietary fats modified the association of PD risk with pesticide exposure. alpha-Linolenic Acid | Fatty Acids | Fatty Acids, Unsaturated | Paraquat | Rotenone Controls for disease:Parkinson Disease | Subjects with disease:Parkinson Disease | Workers United States Details Agricultural Workers' Diseases | Parkinson Disease
19. Vinceti M, et al. (2010). Based on these results, coupled with other epidemiologic data and with findings from animal studies that show specific toxicity of the trace element on motor neurons, we hypothesize that dietary intake of inorganic selenium through drinking water increases the risk for amyotrophic lateral sclerosis. diet Aluminum | Arsenic | Cadmium | Chromium | Copper | Iron | Lead | Lithium | Manganese | Selenic Acid | Zinc Study subjects Italy water, well Aluminum | Arsenic | Cadmium | Chromium | Copper | Iron | Lead | Lithium | Manganese | Selenic Acid | Zinc Details Amyotrophic Lateral Sclerosis
20. Rondeau V, et al. (2009). Aluminum Maladie d'Alzheimer (ALMA+) | Personnes Agees Quid Study (PAQUID) We found that the cognitive decline and the risk of dementia were higher for high consumption of Aluminum from drinking water. age Aluminum | Silicon Dioxide Study subjects France water, drinking, bottled | water, tap Aluminum | Silicon Dioxide Details Alzheimer Disease | cognition
21. Ma L, et al. (2016). We reveal that specific histone modifications are associated with arsenic exposure and oxidative damage, and persistent dysregulation of these histone modifications could be a potential biomarker that evaluates the burden of arsenic exposure and predicts the adverse biological effects. Arsenic Study subjects China hair | lymphocyte | plasma | urine 8-oxo-7-hydrodeoxyguanosine | Arsenic | Malondialdehyde Details Arsenic Poisoning | positive regulation of histone H3-K14 acetylation | positive regulation of histone H3-K18 acetylation | positive regulation of histone H3-K36 trimethylation | positive regulation of histone H3-K9 dimethylation
22. Pi J, et al. (2005). A 13-month arsenic exposure reduction effectively reverses the arsenic-induced impairment of the nitric oxide/cyclic guanosine 3',5'-monophosphate pathway in both males and females and improves peripheral vascular function in males. Arsenic Study subjects China blood | urine | water Arsenic | Cyclic GMP Details Arsenic Poisoning | Vascular Diseases
23. Sampayo-Reyes A, et al. (2010). Altogether, our results evidenced that people living in arsenic-contaminated areas are at risk and that AS3MT genetic variation may play an important role modulating such risk in northern Mexico, especially among children. genetics Arsenic Subjects with gene influence:AS3MT | Children | Study subjects Mexico blood | urine | water, drinking Arsenic | AS3MT Details Arsenic Poisoning | cellular response to DNA damage stimulus
24. Zhang A, et al. (2007). The results showed that long-term Arsenic exposure may be associated with damage of chromosomes and DNA, gene mutations, gene deletions, and alterations of DNA synthesis and repair ability. Arsenic Subjects with disease:Carcinoma | Subjects with disease:Ichthyosis | Subjects with disease:Precancerous Conditions | Study subjects China hair | urine Arsenic Details Arsenic Poisoning | DNA biosynthetic process | DNA repair | gene expression
25. Wasserman GA, et al. (2007). Exposure to arsenic from drinking water was associated with reduced intellectual function before and after adjusting for water manganese for blood lead levels, and for sociodemographic features. diet Arsenic | Lead | Manganese Children Bangladesh blood | urine | water, drinking Arsenic | Lead | Manganese Details Arsenic Poisoning | Learning Disorders
26. Thorpe N, et al. (2005). This hypothesis-generating study explores spatial patterns of childhood cancers in Maryland and investigates their potential associations with herbicides and nitrates in groundwater. Atrazine | metolachlor | Nitrates Children United States Details Bone Neoplasms | Brain Neoplasms | Leukemia | Lymphoma
27. Melzer D, et al. (2010). National Health and Nutrition Examination Survey (NHANES) Higher Bisphenol A (BPA) exposure, reflected in higher urinary concentrations of BPA, is consistently associated with reported heart disease in the general adult population of the USA. bisphenol A Study subjects United States urine bisphenol A Details Angina Pectoris | Coronary Disease | Diabetes Mellitus | Myocardial Infarction | alkaline phosphatase activity | lactate dehydrogenase activity
28. Braun JM, et al. (2009). Health Outcomes and Measures of the Environment (HOME) These results suggest that prenatal bisphenol A exposure may be associated with externalizing behaviors in 2-year-old children, especially among female children. age | sex bisphenol A Children | Pregnant females United States urine bisphenol A Details Neurologic Manifestations | behavior
29. Ciesielski T, et al. (2012). National Health and Nutrition Examination Survey (NHANES) These findings suggest that children who have higher urinary cadmium concentrations may have increased risk of both learning disability and special education. Importantly, we observed these associations at exposure levels that were previously considered to be without adverse effects, and these levels are common among U.S. children. race | socioeconomic status Cadmium Children United States urine Cadmium Details Attention Deficit Disorder with Hyperactivity | Learning Disorders
30. Min JY, et al. (2016). National Health and Nutrition Examination Survey (NHANES) We observed a significant association between blood cadmium levels and Alzheimer disease mortality among older adults in the US. age Cadmium Controls for disease:Diabetes Mellitus | Subjects with disease:Diabetes Mellitus | Controls for disease:Hypertension | Subjects with disease:Hypertension | Study subjects United States blood Cadmium Details Alzheimer Disease
31. Barregard L, et al. (2016). Malmo Diet and Cancer Study Blood cadmium in the highest quartile was associated with incident cardiovascular disease and mortality in our population-based samples of Swedish adults. tobacco Cadmium Study subjects Sweden blood Cadmium Details Cardiovascular Diseases | Myocardial Ischemia | Stroke
32. Peters JL, et al. (2010). National Health and Nutrition Examination Survey (NHANES) Environmental exposure to cadmium was associated with significantly increased stroke and heart failure prevalence. sex | tobacco Cadmium Study subjects United States blood | urine Cadmium Details Heart Failure | Stroke
33. Ferrandiz J, et al. (2004). We used exposure analysis to assess the effect of both protective factors-calcium and magnesium-on mortality from cerebrovascular (ICD-9 430-438) and ischemic heart (ICD-9 410-414) diseases. This study provides statistical evidence of the relationship between mortality from cardiovascular diseases and hardness of drinking water. Calcium | Magnesium Study subjects Spain Details Cardiovascular Diseases | Cerebrovascular Disorders | Death
34. Croxford B, et al. (2008). This study found an association between risk of carbon monoxide exposure at low concentration, and prevalence of self-reported neurological symptoms in the community for those households not in receipt of benefit. Carbon Monoxide Study subjects United Kingdom Details Nervous System Diseases
35. Creswell PD, et al. (2015). Exposure to elevated carbon monoxide levels at an indoor ice arena-Wisconsin, 2014 Carbon Monoxide Study subjects United States air Carbon Monoxide Details Carbon Monoxide Poisoning | Dizziness | Dyspnea | Headache | Nausea | Vomiting
36. Ritz B, et al. (2016). We found ambient air pollution from traffic sources to be associated with risk of Parkinson's Disease, with a 9% higher risk (95% CI: 3, 16.0%) per interquartile range increase (2.97 micrograms per cubic meter) in modeled nitrogen dioxide. Carbon Monoxide | Nitric Oxide | Nitrogen Dioxide Controls for disease:Parkinson Disease | Subjects with disease:Parkinson Disease Denmark Carbon Monoxide | Nitric Oxide | Nitrogen Dioxide Details Parkinson Disease
37. Milojevic A, et al. (2014). This study found no clear evidence for pollution effects on ST-elevation myocardial infarctions and stroke, which ultimately represent thrombogenic processes, though it did for pulmonary embolism. age | sex Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Sulfur Dioxide Study subjects United Kingdom air, ambient Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Sulfur Dioxide Details Arrhythmias, Cardiac | Atrial Fibrillation | Cardiovascular Diseases | Death | Stroke
38. Barbosa SM, et al. (2015). In summary, this study showed that the exposure to air pollution generated mainly from automotive fleets in a large urban center can affect the cardiovascular health of children and may promote a significant health burden on a sensitive group, such as sickle cell patients. Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Sulfur Dioxide Subjects with disease:Anemia, Sickle Cell | Children Brazil air Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Sulfur Dioxide Details Cardiovascular Diseases | Pain
39. Manthripragada AD, et al. (2010). Parkinson's Environment and Genes Study (PEG) The increase in risk we observed among PON1-55 variant carriers for specific organophosphates metabolized by PON1 underscores the importance of considering susceptibility factors when studying environmental exposures in Parkinson disease. genetics Chlorpyrifos | Diazinon | Pesticides Subjects with gene influence:PON1 United States Chlorpyrifos | Diazinon | Pesticides Details Parkinson Disease
40. No authors listed. (2005). This report describes 28 additional cases of hydrogen cyanamide-related illness that occurred during 2002-2004 in Italy; these illnesses suggest that the preventive measures adopted in 2003 to protect agricultural workers are inadequate. Cyanamide | Pesticides Workers Italy Details Agricultural Workers' Diseases | Cardiovascular Diseases | Eye Diseases | Neurologic Manifestations | Poisoning | Signs and Symptoms, Digestive | Signs and Symptoms, Respiratory | Skin Diseases | Skin Manifestations
41. No authors listed. (2001). During Jan-Feb 2001, eight cases of acute illness in the county of Ragusa (Italy) were attributed to exposure to Dormex, a plant growth regulator with hydrogen cyanamide as the active ingredient. Cyanamide | Pesticides Workers Italy Details Agricultural Workers' Diseases | Cardiovascular Diseases | Eye Diseases | Neurologic Manifestations | Poisoning | Signs and Symptoms, Digestive | Skin Diseases | Skin Manifestations
42. Gaspar FW, et al. (2015). Center for the Health Assessment of Mothers and Children of Salinas (CHAMACOS) We conclude that prenatal DDT levels may be associated with delayed processing speed in children at age 7, and the relationship between prenatal DDE levels and children's cognitive development may be modified by sex (with girls being more adversely affected). sex DDT | Dichlorodiphenyl Dichloroethylene Children | Pregnant females United States serum DDT | Dichlorodiphenyl Dichloroethylene Details Neurobehavioral Manifestations | Prenatal Exposure Delayed Effects | cognition
43. Engel SM, et al. (2007). Mount Sinai Children's Environmental Health Study We found an association between both generic and pesticide-specific biomarkers of prenatal organophosphate pesticides and an increased number of abnormal primitive reflexes in neonates, consistent with a previously reported association in a more highly exposed cohort. Dicarboxylic Acids | Dichlorodiphenyl Dichloroethylene | diethyl phosphate | dimethyl phosphate | Organophosphates | Pesticides | Polychlorinated Biphenyls Infants or newborns | Pregnant females United States serum | urine Dicarboxylic Acids | Dichlorodiphenyl Dichloroethylene | diethyl phosphate | dimethyl phosphate | Organophosphates | Polychlorinated Biphenyls Details Prenatal Exposure Delayed Effects | Reflex, Abnormal
44. Tsai RJ, et al. (2014). National Pesticide Information Center (NPIC) | Sentinel Event Notification System for Occupational Risks (SENSOR) Notes from the Field: Acute Illness Associated with Use of Pest Strips - Seven U.S. States and Canada, 2000-2013 Dichlorvos Subjects with disease:Acute Disease Canada|United States Details Acute Disease | Gastrointestinal Diseases | Neurologic Manifestations | Respiratory Tract Diseases
45. Shcherbatykh I, et al. (2005). These results suggest that living near a source of persistent organic pollutants contamination constitutes a risk of exposure and an increased risk of acquiring cerebrovascular disease. age | race | sex Dioxins | Pesticides | Polychlorinated Biphenyls Study subjects United States Details Cerebral Infarction
46. Ton TG, et al. (2010). In this population-based study in which all subjects had HLA DQB1*0602, we found a nearly five-fold increase in narcolepsy risk associated with jobs containing heavy metals and with six or more applications of bug or weed killer. genetics Fertilizers | Metals, Heavy | Pesticides Subjects with gene influence:HLA-DQB1 | Workers United States Details Narcolepsy
47. Yu Y, et al. (2014). Commercial pesticide (occupational exposure) and fertilizer (residential exposure) were both significantly associated with amyotrophic lateral sclerosis in a randomly selected set of patients; these results need to be interpreted cautiously given the small sample size and the lack of direct exposure measures, but they suggest that environmental, occupational, and residential factors warrant close attention in studies examining risk factors for this disease. Fertilizers | Pesticides Controls for disease:Amyotrophic Lateral Sclerosis | Subjects with disease:Amyotrophic Lateral Sclerosis United States Details Amyotrophic Lateral Sclerosis
48. Laidlaw MA, et al. (2015). Legacy petrol lead emissions are associated with increased motor neuron disease death trends in Australia; further examination of the 20 year lag between exposure to petrol lead and onset is warranted. Gasoline Australia air, ambient Lead Details Death | Motor Neuron Disease
49. Bluhm EC, et al. (2007). There was little consistent evidence for an association of synthetic hair dye use with glioma, meningioma, or acoustic neuroma; however, prolonged use of dark-colored permanent dyes warrants further investigation given the high prevalence of hair dyeing. Hair Dyes Subjects with disease:Glioma | Subjects with disease:Meningioma | Subjects with disease:Neuroma, Acoustic United States Details Glioma | Meningioma | Neuroma, Acoustic
50. Louis ED, et al. (2011). Blood harmane and lead concentrations separately correlated with total tremor scores. Participants with high blood concentrations of both toxicants had the highest tremor scores, suggesting an additive effect of these toxicants on tremor severity. harman | Lead Controls for disease:Essential Tremor | Subjects with disease:Essential Tremor United States blood harman | Lead Details Essential Tremor
1–50 of 111 results.