These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Brough HA, et al. (2015).||Consortium of Food Allergy Research Observational Study (CoFAR)||Exposure to peanut antigen in dust through an impaired skin barrier in atopically inflamed skin is a plausible route for peanut skin sensitization and peanut allergy.||diet | disease||Antigens, Plant||Children | Subjects with disease:Dermatitis, Atopic||United States||dust||Antigens, Plant||Details||Peanut Hypersensitivity|
|2.||Kim HH, et al. (2016).||The present study showed that traffic-related and other pollutants around primary school areas are associated with increased risks of allergic diseases among elementary school students.||Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Soot | Sulfur Dioxide||Children||Korea, Republic of||air||Carbon Monoxide | Nitrogen Dioxide | Ozone | Particulate Matter | Soot | Sulfur Dioxide||Details||Asthma | Dermatitis, Atopic | Rhinitis, Allergic|
|3.||Grandjean P, et al. (2010).||These findings suggest that developmental exposure to immunotoxicants may both increase and decrease the risk of allergic disease and that associations between breast-feeding and subsequent allergic disease in children may, at least in part, reflect lactational exposure to immunotoxic food contaminants.||Mercury | Polychlorinated Biphenyls||Children | Mothers | Pregnant females||Faroe Islands||blood | blood, cord | hair | milk, human | serum||Mercury | Polychlorinated Biphenyls||Details||Asthma | Dermatitis, Atopic | immunoglobulin production|