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Disease Neurotoxicity Syndromes

These are exposure studies associated with the disease and all of its children.

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1–14 of 14 results.
  Reference Associated Study Title Author's Summary Study Factors Stressor Receptors Country Medium Exposure Marker Measurements Outcome
1. Mi T, et al. (2013). We describe two cases of acute toxic leukoencephalopathy in migrant workers who were occupationally exposed to organic solvents in construction materials. 1,2,3-trichloropropane | Solvents | Toluene Workers China blood | urine 1,2,3-trichloropropane | Benzoic Acid Details Leukoencephalopathies | Neurotoxicity Syndromes | Occupational Diseases
2. Ma L, et al. (2016). We reveal that specific histone modifications are associated with arsenic exposure and oxidative damage, and persistent dysregulation of these histone modifications could be a potential biomarker that evaluates the burden of arsenic exposure and predicts the adverse biological effects. Arsenic Study subjects China hair | lymphocyte | plasma | urine 8-oxo-7-hydrodeoxyguanosine | Arsenic | Malondialdehyde Details Arsenic Poisoning | positive regulation of histone H3-K14 acetylation | positive regulation of histone H3-K18 acetylation | positive regulation of histone H3-K36 trimethylation | positive regulation of histone H3-K9 dimethylation
3. Sampayo-Reyes A, et al. (2010). Altogether, our results evidenced that people living in arsenic-contaminated areas are at risk and that AS3MT genetic variation may play an important role modulating such risk in northern Mexico, especially among children. genetics Arsenic Subjects with gene influence:AS3MT | Children | Study subjects Mexico blood | urine | water, drinking Arsenic | AS3MT Details Arsenic Poisoning | cellular response to DNA damage stimulus
4. Pi J, et al. (2005). A 13-month arsenic exposure reduction effectively reverses the arsenic-induced impairment of the nitric oxide/cyclic guanosine 3',5'-monophosphate pathway in both males and females and improves peripheral vascular function in males. Arsenic Study subjects China blood | urine | water Arsenic | Cyclic GMP Details Arsenic Poisoning | Vascular Diseases
5. Zhang A, et al. (2007). The results showed that long-term Arsenic exposure may be associated with damage of chromosomes and DNA, gene mutations, gene deletions, and alterations of DNA synthesis and repair ability. Arsenic Subjects with disease:Carcinoma | Subjects with disease:Ichthyosis | Subjects with disease:Precancerous Conditions | Study subjects China hair | urine Arsenic Details Arsenic Poisoning | DNA biosynthetic process | DNA repair | gene expression
6. Wasserman GA, et al. (2007). Exposure to arsenic from drinking water was associated with reduced intellectual function before and after adjusting for water manganese for blood lead levels, and for sociodemographic features. diet Arsenic | Lead | Manganese Children Bangladesh blood | urine | water, drinking Arsenic | Lead | Manganese Details Arsenic Poisoning | Learning Disorders
7. Haefliger P, et al. (2009). Our investigations revealed a mass lead intoxication that occurred through inhalation and ingestion of soil and dust heavily contaminated with lead as a result of informal and unsafe used lead-acid batteries recycling. Lead Children | Subjects with disease:Lead Poisoning | Study subjects Senegal blood Lead Details Death | Lead Poisoning, Nervous System, Childhood
8. Rajan P, et al. (2007). Normative Aging Study (NAS) We observed a deleterious association between current and cumulative lead burden and risk of psychiatric symptoms. At a given cumulative lead burden, participants with the ALAD 1-1 genotype appeared to be at greater risk than participants who were carriers of at least one variant ALAD allele. genetics Lead Subjects with gene influence:ALAD | Veterans United States blood | bone | patella | tibia Lead Details Lead Poisoning, Nervous System | Mental Disorders
9. Cecil KM, et al. (2011). Cincinnati Lead Study Gray-matter N-acetyl aspartate reductions associated with increasing childhood blood lead levels suggest that sustained childhood lead exposure produces an irreversible pattern of neuronal dysfunction, whereas associated white-matter choline declines indicate a permanent alteration to myelin architecture. Lead Study subjects United States Lead Details Lead Poisoning, Nervous System | metabolic process
10. Palaniappan K, et al. (2011). Among urban Indian children, higher blood lead levels are associated with decreased visual-motor abilities, particularly visual-motor integration. Lead Children India blood Lead Details Lead Poisoning, Nervous System, Childhood | behavior
11. Kulkarni PA, et al. (2015). Severe illness from methyl bromide exposure at a condominium resort - U.S. Virgin Islands, March 2015 methyl bromide Study subjects Virgin Islands, U.S. serum methyl bromide Details Neurologic Manifestations | Neurotoxicity Syndromes
12. Yokoo EM, et al. (2003). This study suggests that adults exposed to methylmercury may be at risk for deficits in neurocognitive function. Methylmercury Compounds Study subjects Brazil hair Methylmercury Compounds Details Cognition Disorders | Mercury Poisoning, Nervous System | cognition
13. Winnik L, et al. (2004). The aim of this study was the presentation of the chronic tetraethyl lead poisoning of a four-person family. Tetraethyl Lead Children | Subjects with disease:Hallucinations | Study subjects Poland serum Lead Details Lead Poisoning, Nervous System, Adult
14. No authors listed. (2003). This report describes the first known case in the United States of exposure to a Chinese rodenticide containing the toxin tetramethylenedisulfotetramine (TETS), a convulsant poison. tetramethylenedisulfotetramine Infants or newborns United States Details Neurotoxicity Syndromes
1–14 of 14 results.