These are exposure studies associated with the disease and all of its children.
|Reference||Associated Study Title||Author's Summary||Study Factors||Stressor||Receptors||Country||Medium||Exposure Marker||Measurements||Outcome|
|1.||Shcherbatykh I, et al. (2005).||These results suggest that living near a source of persistent organic pollutants contamination constitutes a risk of exposure and an increased risk of acquiring cerebrovascular disease.||age | race | sex||Dioxins | Pesticides | Polychlorinated Biphenyls||Study subjects||United States||Details||Cerebral Infarction|
|2.||Mao X, et al. (2014).||We describe associations among basic characteristics, seasons, and diseases with serum levels of 25-hydroxyvitamin D in patients and note that 25(OH)D3 levels are low while 25(OH)D2 levels are high among patients with lung diseases, dyskinesias, and coronary heart disease, and participants with diabetes and cerebral infarction have higher 25(OH)D3 serum concentrations compared with lung disease patients.||age | disease||Vitamin D||Subjects with disease:Cerebral Infarction | Subjects with disease:Coronary Disease | Subjects with disease:Diabetes Mellitus | Subjects with disease:Dyskinesias | Subjects with disease:Hypertension | Subjects with disease:Lung Diseases | Study subjects||China||serum||25-Hydroxyvitamin D 2 | 25-hydroxyvitamin D | Calcifediol||Details|
|3.||Majumdar V, et al. (2015).||The relationship between vitamin D deficiency and stroke was cross-sectionally evaluated in the high-risk Asian Indian population, and we conclude that hypertension partly explains the association between low 25-hydroxyvitamin D levels and ischemic stroke.||disease||Vitamin D||Controls for disease:Cerebral Infarction | Subjects with disease:Cerebral Infarction | Subjects with disease:Hypertension||India||serum||25-hydroxyvitamin D||Details||Cerebral Infarction|